ac devd pna substrate assay kit Search Results


ac-devd-cho  (Promega)
90
Promega ac-devd-cho
Ac Devd Cho, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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acetyl asp-glu-val-asp 7-amido-4-methylcoumarin (ac-devd-amc)  (Enzo Biochem)
90
Enzo Biochem acetyl asp-glu-val-asp 7-amido-4-methylcoumarin (ac-devd-amc)
Acetyl Asp Glu Val Asp 7 Amido 4 Methylcoumarin (Ac Devd Amc), supplied by Enzo Biochem, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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n acetyl ac asp glu val asp p nitroanilide  (Beyotime)
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Beyotime n acetyl ac asp glu val asp p nitroanilide
N Acetyl Ac Asp Glu Val Asp P Nitroanilide, supplied by Beyotime, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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caspase-3 cellular assay kit plus with ac-devd- p -nitroanilide  (Biomol GmbH)
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Biomol GmbH caspase-3 cellular assay kit plus with ac-devd- p -nitroanilide
Caspase 3 Cellular Assay Kit Plus With Ac Devd P Nitroanilide, supplied by Biomol GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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caspase 8 activity assay kit  (Beyotime)
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Beyotime caspase 8 activity assay kit
Caspase 8 Activity Assay Kit, supplied by Beyotime, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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caspase 3 inhibitor  (Danaher Inc)
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Danaher Inc caspase 3 inhibitor
Caspase 3 Inhibitor, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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caspase kit ac-dev-afc/z-vad-fmk  (Geno Tech Inc)
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Geno Tech Inc caspase kit ac-dev-afc/z-vad-fmk
Caspase Kit Ac Dev Afc/Z Vad Fmk, supplied by Geno Tech Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ac devd pna  (Elabscience Biotechnology)
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Elabscience Biotechnology ac devd pna
Ac Devd Pna, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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devd-pna  (Becton Dickinson)
90
Becton Dickinson devd-pna
A 4 hr reoxygenation response is seen as changes in brain Glut3 (G3) and Glut1 (G1) noted as an effect of genotype (F=231.17, p<0.001), treatment-exposure (F=8.24, p<0.001) and genotype X treatment-exposure (F=5.20, p=0.003) on Glut3 (A). A genotype effect in Nx-Crtl and a treatment-exposure effect of Hx-Crtl and Hx-Ipsi is observed on Glut1 (Fisher’s PLSD Op<0.05) (A). Mitochondrial apoptotic proteins, Bax and Bcl2 (Fisher’s PLSD Op<0.05) demonstrate an effect of genotype (F=16.35 or 25.76, p<0.001) but not that of treatment-exposure or genotype X treatment-exposure (B). A 24 hr response of brain Glut3 (G3) is seen as genotype (F=192.25, p<0.001) and treatment-exposure (F=4.99, p=0.004) effects alone and Glut1 (G1) is also seen as genotype (F=10.84, p=0.002) and treatment-exposure (F=3.61, p=0.02) effects alone (C). Bax (genotype – F=8.10, p=0.007; genotype X treatment-exposure – F=9.93, p<0.001) and Bcl2 (genotype X treatment-exposure – F=2.98, p=0.04) (D), <t>pro-caspase-3</t> (genotype – F=67.52, p<0.001; treatment-exposure – F=8.26, p<0.001) (E) and caspase-3 enzyme activity (genotype – F=13.22, p<0.001; treatment-exposure – F=18.04, p<0.001) (F) in Nx-Crtl, Nx-Ipsi, Hx-Crtl and Hx-Ipsi in WT and glut3+/− mice are seen. In all panels representative Western blots are shown in the inset with vinculin (vin) or β-actin as internal controls and the quantification depicted below. N=7 per genotype per experimental group (Hx vs Nx) and per exposure (ischemic vs non-ischemic side) for all measurements. † or *Holm-Sidak test following two-way ANOVA or OFisher’s PLSD test following one-way ANOVA demonstrates inter-group differences.
Devd Pna, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
devd-pna - by Bioz Stars, 2026-07
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caspase 3 assay kit  (Becton Dickinson)
90
Becton Dickinson caspase 3 assay kit
A 4 hr reoxygenation response is seen as changes in brain Glut3 (G3) and Glut1 (G1) noted as an effect of genotype (F=231.17, p<0.001), treatment-exposure (F=8.24, p<0.001) and genotype X treatment-exposure (F=5.20, p=0.003) on Glut3 (A). A genotype effect in Nx-Crtl and a treatment-exposure effect of Hx-Crtl and Hx-Ipsi is observed on Glut1 (Fisher’s PLSD Op<0.05) (A). Mitochondrial apoptotic proteins, Bax and Bcl2 (Fisher’s PLSD Op<0.05) demonstrate an effect of genotype (F=16.35 or 25.76, p<0.001) but not that of treatment-exposure or genotype X treatment-exposure (B). A 24 hr response of brain Glut3 (G3) is seen as genotype (F=192.25, p<0.001) and treatment-exposure (F=4.99, p=0.004) effects alone and Glut1 (G1) is also seen as genotype (F=10.84, p=0.002) and treatment-exposure (F=3.61, p=0.02) effects alone (C). Bax (genotype – F=8.10, p=0.007; genotype X treatment-exposure – F=9.93, p<0.001) and Bcl2 (genotype X treatment-exposure – F=2.98, p=0.04) (D), <t>pro-caspase-3</t> (genotype – F=67.52, p<0.001; treatment-exposure – F=8.26, p<0.001) (E) and caspase-3 enzyme activity (genotype – F=13.22, p<0.001; treatment-exposure – F=18.04, p<0.001) (F) in Nx-Crtl, Nx-Ipsi, Hx-Crtl and Hx-Ipsi in WT and glut3+/− mice are seen. In all panels representative Western blots are shown in the inset with vinculin (vin) or β-actin as internal controls and the quantification depicted below. N=7 per genotype per experimental group (Hx vs Nx) and per exposure (ischemic vs non-ischemic side) for all measurements. † or *Holm-Sidak test following two-way ANOVA or OFisher’s PLSD test following one-way ANOVA demonstrates inter-group differences.
Caspase 3 Assay Kit, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/ac+devd+pna+substrate+assay+kit/pm33398876-53-12-17?v=Becton+Dickinson
Average 90 stars, based on 1 article reviews
caspase 3 assay kit - by Bioz Stars, 2026-07
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ac-devd-amc protease assay kit  (Becton Dickinson)
90
Becton Dickinson ac-devd-amc protease assay kit
A 4 hr reoxygenation response is seen as changes in brain Glut3 (G3) and Glut1 (G1) noted as an effect of genotype (F=231.17, p<0.001), treatment-exposure (F=8.24, p<0.001) and genotype X treatment-exposure (F=5.20, p=0.003) on Glut3 (A). A genotype effect in Nx-Crtl and a treatment-exposure effect of Hx-Crtl and Hx-Ipsi is observed on Glut1 (Fisher’s PLSD Op<0.05) (A). Mitochondrial apoptotic proteins, Bax and Bcl2 (Fisher’s PLSD Op<0.05) demonstrate an effect of genotype (F=16.35 or 25.76, p<0.001) but not that of treatment-exposure or genotype X treatment-exposure (B). A 24 hr response of brain Glut3 (G3) is seen as genotype (F=192.25, p<0.001) and treatment-exposure (F=4.99, p=0.004) effects alone and Glut1 (G1) is also seen as genotype (F=10.84, p=0.002) and treatment-exposure (F=3.61, p=0.02) effects alone (C). Bax (genotype – F=8.10, p=0.007; genotype X treatment-exposure – F=9.93, p<0.001) and Bcl2 (genotype X treatment-exposure – F=2.98, p=0.04) (D), <t>pro-caspase-3</t> (genotype – F=67.52, p<0.001; treatment-exposure – F=8.26, p<0.001) (E) and caspase-3 enzyme activity (genotype – F=13.22, p<0.001; treatment-exposure – F=18.04, p<0.001) (F) in Nx-Crtl, Nx-Ipsi, Hx-Crtl and Hx-Ipsi in WT and glut3+/− mice are seen. In all panels representative Western blots are shown in the inset with vinculin (vin) or β-actin as internal controls and the quantification depicted below. N=7 per genotype per experimental group (Hx vs Nx) and per exposure (ischemic vs non-ischemic side) for all measurements. † or *Holm-Sidak test following two-way ANOVA or OFisher’s PLSD test following one-way ANOVA demonstrates inter-group differences.
Ac Devd Amc Protease Assay Kit, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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apo-assay kit containing cpp32 specific peptide, ac-dev-afc, and cpp32 inhibitor, zvad-fmk, + lysing buffer  (Geno Tech Inc)
90
Geno Tech Inc apo-assay kit containing cpp32 specific peptide, ac-dev-afc, and cpp32 inhibitor, zvad-fmk, + lysing buffer
A 4 hr reoxygenation response is seen as changes in brain Glut3 (G3) and Glut1 (G1) noted as an effect of genotype (F=231.17, p<0.001), treatment-exposure (F=8.24, p<0.001) and genotype X treatment-exposure (F=5.20, p=0.003) on Glut3 (A). A genotype effect in Nx-Crtl and a treatment-exposure effect of Hx-Crtl and Hx-Ipsi is observed on Glut1 (Fisher’s PLSD Op<0.05) (A). Mitochondrial apoptotic proteins, Bax and Bcl2 (Fisher’s PLSD Op<0.05) demonstrate an effect of genotype (F=16.35 or 25.76, p<0.001) but not that of treatment-exposure or genotype X treatment-exposure (B). A 24 hr response of brain Glut3 (G3) is seen as genotype (F=192.25, p<0.001) and treatment-exposure (F=4.99, p=0.004) effects alone and Glut1 (G1) is also seen as genotype (F=10.84, p=0.002) and treatment-exposure (F=3.61, p=0.02) effects alone (C). Bax (genotype – F=8.10, p=0.007; genotype X treatment-exposure – F=9.93, p<0.001) and Bcl2 (genotype X treatment-exposure – F=2.98, p=0.04) (D), <t>pro-caspase-3</t> (genotype – F=67.52, p<0.001; treatment-exposure – F=8.26, p<0.001) (E) and caspase-3 enzyme activity (genotype – F=13.22, p<0.001; treatment-exposure – F=18.04, p<0.001) (F) in Nx-Crtl, Nx-Ipsi, Hx-Crtl and Hx-Ipsi in WT and glut3+/− mice are seen. In all panels representative Western blots are shown in the inset with vinculin (vin) or β-actin as internal controls and the quantification depicted below. N=7 per genotype per experimental group (Hx vs Nx) and per exposure (ischemic vs non-ischemic side) for all measurements. † or *Holm-Sidak test following two-way ANOVA or OFisher’s PLSD test following one-way ANOVA demonstrates inter-group differences.
Apo Assay Kit Containing Cpp32 Specific Peptide, Ac Dev Afc, And Cpp32 Inhibitor, Zvad Fmk, + Lysing Buffer, supplied by Geno Tech Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/ac+devd+pna+substrate+assay+kit/pmc01343545-89-15-30?v=Geno+Tech+Inc
Average 90 stars, based on 1 article reviews
apo-assay kit containing cpp32 specific peptide, ac-dev-afc, and cpp32 inhibitor, zvad-fmk, + lysing buffer - by Bioz Stars, 2026-07
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Image Search Results


A 4 hr reoxygenation response is seen as changes in brain Glut3 (G3) and Glut1 (G1) noted as an effect of genotype (F=231.17, p<0.001), treatment-exposure (F=8.24, p<0.001) and genotype X treatment-exposure (F=5.20, p=0.003) on Glut3 (A). A genotype effect in Nx-Crtl and a treatment-exposure effect of Hx-Crtl and Hx-Ipsi is observed on Glut1 (Fisher’s PLSD Op<0.05) (A). Mitochondrial apoptotic proteins, Bax and Bcl2 (Fisher’s PLSD Op<0.05) demonstrate an effect of genotype (F=16.35 or 25.76, p<0.001) but not that of treatment-exposure or genotype X treatment-exposure (B). A 24 hr response of brain Glut3 (G3) is seen as genotype (F=192.25, p<0.001) and treatment-exposure (F=4.99, p=0.004) effects alone and Glut1 (G1) is also seen as genotype (F=10.84, p=0.002) and treatment-exposure (F=3.61, p=0.02) effects alone (C). Bax (genotype – F=8.10, p=0.007; genotype X treatment-exposure – F=9.93, p<0.001) and Bcl2 (genotype X treatment-exposure – F=2.98, p=0.04) (D), pro-caspase-3 (genotype – F=67.52, p<0.001; treatment-exposure – F=8.26, p<0.001) (E) and caspase-3 enzyme activity (genotype – F=13.22, p<0.001; treatment-exposure – F=18.04, p<0.001) (F) in Nx-Crtl, Nx-Ipsi, Hx-Crtl and Hx-Ipsi in WT and glut3+/− mice are seen. In all panels representative Western blots are shown in the inset with vinculin (vin) or β-actin as internal controls and the quantification depicted below. N=7 per genotype per experimental group (Hx vs Nx) and per exposure (ischemic vs non-ischemic side) for all measurements. † or *Holm-Sidak test following two-way ANOVA or OFisher’s PLSD test following one-way ANOVA demonstrates inter-group differences.

Journal: Journal of neuroscience research

Article Title: Hypoxic-Ischemic Brain Injury Exacerbates Neuronal Apoptosis and Precipitates Spontaneous Seizures in Glucose Transporter Isoform 3 Heterozygous Null Mice

doi: 10.1002/jnr.22487

Figure Lengend Snippet: A 4 hr reoxygenation response is seen as changes in brain Glut3 (G3) and Glut1 (G1) noted as an effect of genotype (F=231.17, p<0.001), treatment-exposure (F=8.24, p<0.001) and genotype X treatment-exposure (F=5.20, p=0.003) on Glut3 (A). A genotype effect in Nx-Crtl and a treatment-exposure effect of Hx-Crtl and Hx-Ipsi is observed on Glut1 (Fisher’s PLSD Op<0.05) (A). Mitochondrial apoptotic proteins, Bax and Bcl2 (Fisher’s PLSD Op<0.05) demonstrate an effect of genotype (F=16.35 or 25.76, p<0.001) but not that of treatment-exposure or genotype X treatment-exposure (B). A 24 hr response of brain Glut3 (G3) is seen as genotype (F=192.25, p<0.001) and treatment-exposure (F=4.99, p=0.004) effects alone and Glut1 (G1) is also seen as genotype (F=10.84, p=0.002) and treatment-exposure (F=3.61, p=0.02) effects alone (C). Bax (genotype – F=8.10, p=0.007; genotype X treatment-exposure – F=9.93, p<0.001) and Bcl2 (genotype X treatment-exposure – F=2.98, p=0.04) (D), pro-caspase-3 (genotype – F=67.52, p<0.001; treatment-exposure – F=8.26, p<0.001) (E) and caspase-3 enzyme activity (genotype – F=13.22, p<0.001; treatment-exposure – F=18.04, p<0.001) (F) in Nx-Crtl, Nx-Ipsi, Hx-Crtl and Hx-Ipsi in WT and glut3+/− mice are seen. In all panels representative Western blots are shown in the inset with vinculin (vin) or β-actin as internal controls and the quantification depicted below. N=7 per genotype per experimental group (Hx vs Nx) and per exposure (ischemic vs non-ischemic side) for all measurements. † or *Holm-Sidak test following two-way ANOVA or OFisher’s PLSD test following one-way ANOVA demonstrates inter-group differences.

Article Snippet: Subsequently the chromophore p-nitroaniline (pNA) was detected spectrophotometrically (405 nm) after its cleavage by endogenous caspase-3 from the labeled caspase-specific substrate, DEVD-pNA (ApoAlert Caspase Colorimetric Assay Kit; BD Biosciences Clontech, Palo Alto, CA).

Techniques: Activity Assay, Western Blot